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Tumor cells interact with immune system elements that are functioning to impede the proliferation and metastasis of melanoma throughout the melanomagenesis process

Natural variation in the activity of these enzymes can contribute to a range of drug responses and side effects on an individual basis

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plasmin derived from the reactive brain stroma cleaves Fas ligand (FasL) on the surface of astrocytes, mobilising it to interact with its receptor, Fas, to induce apoptosis in tumour cells