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(2001) [86] reported that L-Carnitine, acting as a cofactor in fatty acid transport, may have a reparative effect by eliminating elevated intracellular levels of toxic acetyl-coenzyme A or by replacing fatty acids in the phospholipid membrane

By using macrophage and mouse infection models, we concluded that the endogenous NO promotes bacterial survival by activating host cellular autophagy during bacterial infection

As to pharmacologic effects, the hydroalcoholic extract from the bark of Abarema cochliacarpos showed antimicrobial activity

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