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The ability of autophagy to selectively target intracellular pathogens for destruction is now considered a vital aspect of the innate immune response (Levine and Kroemer, 2019)

The hypothesis has expanded to include vascular dysfunction, oxidative stress, microglial activation, and impaired proteolysis (Roda et al., 2022)

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[] There is controversy about the liver to the skeletal muscle crosstalk in patients with comorbidity of NAFLD and sarcopenia, as that NAFLD and sarcopenia share many complex interplaying mechanisms, including insulin resistance and myostatin and adiponectin homeostasis, which make it difficult to determine the actual cause, therefore, identifying whether sarcopenia is a cause, consequence, both, or neither of fatty liver is challenging