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Olaparib, a PARP inhibitor (PARPi), has been demonstrated to not only exacerbate cellular senescence by amplifying DNA damage, upregulating senescence markers (e.g., SA--gal and p21), and inducing irreversible cell cycle arrest, particularly in replicatively senescent macrophages but also trigger mixed apoptotic and necrotic cell death at high doses

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Improvements in liver enzymes such as alanine aminotransferase (ALT) and aspartate aminotransferase (AST) as well as fibrosis markers such as the NAFLD fibrosis score and hepatic steatosis index, have been reported

Disruptions in the coordinated temporal pattern of Flt3L production during development significantly affect the balance between pDCs and Tregs, increasing the vulnerability of newborns to sLRIs and the subsequent development of immunopathology associated with asthma

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Specifically, in ischemic/high-ROS environments, BACH1 tends to be degraded and acts to suppress angiogenesis by inhibiting the Wnt/-catenin signaling pathway and recruiting HDAC1, among other mechanisms